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Iated immune [58] responses . Subviral particles, genetically engineered plant viruses, insect-derived virus-likeIated immune [58] responses . Subviral particles, genetically engineered plant viruses, insect-derived virus-like particles, are suitable as presentation scaffold and adjuvant platform for multimeric display of foreign antigens in a correct, ordered and highly repetitive three-dim
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Hospital centers in Alberta, Canada. Standard phase sensitive inversion recovery sequences were used on commercially available scanners (Siemens Avanto and Sonata, Siemens, Erlangen, Germany). Delayed enhancement (DE) was evaluated visually using CMR42 (Circle Canada Inc, Calgary, Canada) software analysis package by two independent readers. DE had to be cross-referenced in two orthogonal views. D
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Osclerotic plaque volume in the superficial femoral artery when compared to those already treated with statin given ezetimibe. Reverse vessel wall remodeling was noted among PAD patients newly treated with statins compared to those with ezetimibe added to preexisting statin therapy. Thus the degree and/or mechanism of LDL lowering rather than the final LDL achieved may be important in halting athe
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Volakas white marble is from Greece well-known white marble quarry. Volakas could be cut to tiles, slabs, floor, wall cladding, fire in polish, hone. Volakas white marble is off white marble.
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Volakas white marble is from Greece renowned white marble quarry. Volakas could be minimize to tiles, slabs, ground, wall cladding, fireplace in polish, hone. Volakas white marble is off white marble.
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And Q506P), chosen to represent the spectrum of disease-associated PTPN11 mutations. Mutants D61G, E139D, and N308D are found in NS, Y279C is a canonical LS mutation [16,21,42], and Q506P has been reported in both disorders [16], although it is unclear whether this reflects misdiagnosis or true bipotentiality of this allele. The D61G mutation affects the N-SH2 domain, E139D lies within the C-SH2 d
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And Q506P), chosen to represent the spectrum of disease-associated PTPN11 mutations. Mutants D61G, E139D, and N308D are found in NS, Y279C is a canonical LS mutation [16,21,42], and Q506P has been reported in both disorders [16], although it is unclear whether this reflects misdiagnosis or true bipotentiality of this allele. The D61G mutation affects the N-SH2 domain, E139D lies within the C-SH2 d